Seminars & Events
Friday, October 4, 2013: Dr. Laurie Ryan, SMCM '86 (National Institute on Aging) will speak on "Alzheimer's Disease: Targets and Treatments" at 3:00 pm in Goodpaster Hall 195.
Monday, October 21, 2013: Dr. Greg Elmer (University of Maryland Baltimore) will speak on "Domains and Constructs in Motivation: Where Does the Habenula Fit In?" at 4:45 pm in Goodpaster Hall 195.
Friday, October 25, 2013: Dr. Terry Davidson (American University) will speak on "Why We Overeat and Become Obese? It Could be What We Think!" at 3:00 pm in Goodpaster Hall 195.
Dr. Gwen Calhoon '06 recently received her Ph.D. in Neuroscience from the University of Maryland Baltimore, and was inducted into Nu Rho Psi.
Kircher, Daniel (2010). Perinatal Exposure to Bisphenol-A: Does it Produce Similar Behavioral Deficits to Those Observed in Animal Models of Schizophrenia? Mentor: Dr. Anne Marie Brady
The endocrine disrupting chemical Bisphenol-A (BPA) has the ability to cross the placental barrier and produce changes in offspring. The chemical has been found to cause disruptions of dopamine systems. Disruptions of dopamine observed in BPA treated animals have also been observed in animal models of schizophrenia. We examined the effects of perinatal treatment of BPA on pre-pulse inhibition; novelty induced hyperlocomotion, the Morris water maze (MWM), and social interaction and Dopamine (DA) cell numbers in the substantia nigra of adult rats. BPA treated animals had decreased time spent in the target quadrant in the MWM and had increased distance traveled in the open field. BPA treated animals also had decreases in the time spent sniffing/grooming partners. BPA treatment however, did not disrupt DA cell numbers. These findings could indicate a connection between perinatal BPA exposure and animal models of schizophrenia.