Seminars & Events

Thursday, September 11, 2014: Dr. Bevil Conway (Wellesley College) will speak on his research in visual neuroscience and color at 4:30 pm in Goodpaster Hall 195.

Monday, October 27, 2014: Dr. Todd Gould (University of Maryland Baltimore) will speak on "Genes to behaviors to treatments in bipolar disorder" at 4:45 pm in Goodpaster Hall 195

Friday, December 5, 2014:  Dr. Brian Mathur (University of Maryland Baltimore) will speak on "Braking bad: Aberrant inhibitory neurotransmission in addiction" at 3:00 pm in Goodpaster Hall 195.


Alumni Highlight

Check out Jordan Gaines Lewis '11's award-winning blog, Gaines on Brains.




SMP Spotlight

Katie Gluskin and Jeff Haus present their SMP
Katie Gluskin and Jeff Haus, "Entorhinal Cortex Lesions, Habituation, and Latent Inhibition," 2013. Gluskin and Haus, the 2013 co-winners of the Neuroscience Award, infused a neurotoxin into the entorhinal cortex of rats to induce a lesion, and measured the resulting habituation and latent inhibition behavior within a fear conditioning paradigm.


Kircher, Daniel (2010).   Perinatal Exposure to Bisphenol-A: Does it Produce Similar Behavioral Deficits to Those Observed in Animal Models of Schizophrenia?  Mentor: Dr. Anne Marie Brady


The endocrine disrupting chemical Bisphenol-A (BPA) has the ability to cross the placental barrier and produce changes in offspring. The chemical has been found to cause disruptions of dopamine systems. Disruptions of dopamine observed in BPA treated animals have also been observed in animal models of schizophrenia. We examined the effects of perinatal treatment of BPA on pre-pulse inhibition; novelty induced hyperlocomotion, the Morris water maze (MWM), and social interaction and Dopamine (DA) cell numbers in the substantia nigra of adult rats. BPA treated animals had decreased time spent in the target quadrant in the MWM and had increased distance traveled in the open field. BPA treated animals also had decreases in the time spent sniffing/grooming partners. BPA treatment however, did not disrupt DA cell numbers.  These findings could indicate a connection between perinatal BPA exposure and animal models of schizophrenia.