Seminars & Events

Thursday, September 11, 2014: Dr. Bevil Conway (Wellesley College) will speak on his research in visual neuroscience and color at 4:30 pm in Goodpaster Hall 195.

Monday, October 27, 2014: Dr. Todd Gould (University of Maryland Baltimore) will speak on "Genes to behaviors to treatments in bipolar disorder" at 4:45 pm in Goodpaster Hall 195

Friday, December 5, 2014:  Dr. Brian Mathur (University of Maryland Baltimore) will speak on "Braking bad: Aberrant inhibitory neurotransmission in addiction" at 3:00 pm in Goodpaster Hall 195.


Alumni Highlight

Check out Jordan Gaines Lewis '11's award-winning blog, Gaines on Brains.




SMP Spotlight

Katie Gluskin and Jeff Haus present their SMP
Katie Gluskin and Jeff Haus, "Entorhinal Cortex Lesions, Habituation, and Latent Inhibition," 2013. Gluskin and Haus, the 2013 co-winners of the Neuroscience Award, infused a neurotoxin into the entorhinal cortex of rats to induce a lesion, and measured the resulting habituation and latent inhibition behavior within a fear conditioning paradigm.


Dippel, W. Chris (2012).  Cognitive and behavioral impairments evaluated in the neonatal ventral hippocampus lesion rat model of schizophrenia. (Mentor: A.M. Brady)


The neonatal ventral hippocampus lesion (NVHL) is a proposed rat model for schizophrenia. The present study assessed the effects of the NVHL in social interaction, pre-pulse inhibition, spontaneous locomotion, response to amphetamine, set-shifting, and reversal learning. Rats with an NVHL showed deficits in pre-pulse inhibition, and set-shifting as well as a stronger response to amphetamine. Few effects were found in social interaction, and there was no effect during reversal learning. Pre-exposed rats made more perseverative errors during the set-shift task suggesting the NVHL causes impairments in PFC functioning. Percent pre-pulse inhibition was negatively correlated with trials to criterion on the shift day of the set-shifting task, response to amphetamine, and spontaneous locomotion suggesting there may be a relationship between behavior deficits elicited by the NVHL.